Copper Peptide โ 50mg/vial
$79 AUD
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GHK-Cu (Glycyl-L-histidyl-L-lysine Copper) is a naturally occurring copper tripeptide complex that was first isolated from human plasma in 1973 by Dr. Loren Pickart at the University of Washington. Remarkably, GHK-Cu levels decline with age โ the plasma of a 20-year-old contains significantly higher levels than that of a 60-year-old โ making it one of the most studied peptides in aging and tissue regeneration research.
Preclinical research has demonstrated GHK-Cu's ability to stimulate collagen synthesis, promote angiogenesis, modulate inflammation, and support tissue remodelling. It is one of the most extensively researched copper peptides, with hundreds of published studies spanning wound healing, dermal research, hair follicle studies, and tissue regeneration. Its well-characterised safety profile and multi-pathway mechanism make it a cornerstone compound in regenerative research.
"FOR RESEARCH USE ONLY. This information is provided for scientific and educational purposes only. It does not constitute medical advice or a recommendation for human use."
| Compound | GHK-Cu |
| Form | Lyophilized powder |
| Dosage | 50mg/vial |
| Purity | 99%+ (HPLC verified) |
| Testing | Third-party HPLC + MS |
| Storage | 2-8ยฐC (refrigerated) |
| Origin | GMP facility |
GHK-Cu exerts its biological effects through multiple pathways. Its primary mechanism involves copper ion delivery to copper-dependent enzymes, including superoxide dismutase (SOD), cytochrome c oxidase, and lysyl oxidase โ each playing a critical role in antioxidant defence, cellular energy production, and collagen cross-linking respectively.
Research demonstrates that GHK-Cu upregulates collagen synthesis by activating transforming growth factor beta (TGF-ฮฒ) signalling and increasing fibroblast proliferation. It also promotes angiogenesis through vascular endothelial growth factor (VEGF) upregulation, supports tissue remodelling via matrix metalloproteinase (MMP) modulation, and exhibits anti-inflammatory activity by suppressing TNF-ฮฑ and IL-1ฮฒ. Its ability to act as a copper ionophore โ transporting copper into cells where it's needed โ is central to its broad range of reported biological activities.
Every batch independently tested via HPLC and Mass Spectrometry. COAs published on site.
Based in Australia with express shipping nationwide. Discreet, temperature-controlled packaging.
Every batch independently tested via HPLC and Mass Spectrometry. COAs published on site with full batch details.
GHK-Cu functions primarily as a copper ionophore โ delivering copper ions to copper-dependent enzymes including superoxide dismutase (antioxidant defence), cytochrome c oxidase (cellular energy), and lysyl oxidase (collagen cross-linking). It also activates TGF-ฮฒ signalling to stimulate collagen synthesis, upregulates VEGF for angiogenesis, and modulates inflammatory cytokines including TNF-ฮฑ and IL-1ฮฒ.
GHK-Cu is unique among copper peptides in that it is a naturally occurring human tripeptide โ first isolated from human plasma. Its specific sequence (glycyl-histidyl-lysine) gives it high affinity for copper binding and specific biological activities that synthetic copper peptides do not fully replicate. Its levels naturally decline with age, making it particularly relevant for aging research.
GHK-Cu is water-soluble and reconstituted with sterile bacteriostatic water. The peptide has a characteristic blue colour when in solution (due to the copper complex). It should be stored lyophilized at 2-8ยฐC and, once reconstituted, used within 7-14 days. Standard research concentrations range from 1-100 ยตM depending on the experimental model. Always follow your institution's ethical guidelines.
GHK-Cu is legal to purchase for legitimate research purposes in Australia. It is not a scheduled substance when intended for research use only. All sales are strictly for laboratory research purposes โ not for human or veterinary consumption.
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